Skip Navigation

QJM 2008 101(11):829-830; doi:10.1093/qjmed/hcn141
This Article
Right arrow Full Text Freely available
Right arrow FREE Full Text (PDF) Freely available
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by Bannon, M.
Right arrow Search for Related Content
PubMed
Right arrow Articles by Bannon, M.
Social Bookmarking
 Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

© The Author 2008. Published by Oxford University Press on behalf of the Association of Physicians. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

Elements: In this month's issue

The first 10% of the full text of this article appears below.

The review by Sánchez-Quiñones et al discusses the complex relationship between atrial fibrillation (AF) and the use of statins, a subject area that has attracted media attention this year. The suppression by statins of cholesterol biosynthesis is well documented. However it is now speculated that statins also have anti-inflammatory properties. The pathogenesis of AF is multifactorial but there is now some evidence to support the hypothesis that oxidative stress and inflammation may play a role in its development. Unsurprisingly therefore, . . . [Full Text of this Article]

Michael Bannon

Editor, QJM


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?