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QJM 2005 98(6):463-464; doi:10.1093/qjmed/hci075
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© The Author 2005. Published by Oxford University Press on behalf of the Association of Physicians. All rights reserved. For Permissions, please email: journals.permissions@oupjournals.org

Correspondence

Post-traumatic hyponatraemia due to acute hypopituitarism

The first 10% of the full text of this article appears below.

Sir,

Hyponatraemia following traumatic brain injury (TBI) is a common complication, occurring in 13% of cases.1 The commonest cause of hyponatraemia is the syndrome of inappropriate antidiuretic hormone secretion (SIADH), which is responsible for over 90% of cases,1 whereas cerebral salt wasting, medications and injudicious use of intravenous fluids may also cause hyponatraemia following TBI. As glucocorticoid deficiency can present . . . [Full Text of this Article]

A. Agha, M. Sherlock and C.J. Thompson

Department of Endocrinology Beaumont Hospital Dublin Ireland e-mail: amaragha@yahoo.com


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H. J. Schneider, I. Kreitschmann-Andermahr, E. Ghigo, G. K. Stalla, and A. Agha
Hypothalamopituitary Dysfunction Following Traumatic Brain Injury and Aneurysmal Subarachnoid Hemorrhage: A Systematic Review
JAMA, September 26, 2007; 298(12): 1429 - 1438.
[Abstract] [Full Text] [PDF]